ACTUALIZACIÓN EN EL DIAGNÓSTICO Y TRATAMIENTO DE LA EPILEPSIA
La siguiente es una revisión de la epilepsia, su clasificación, fisiopatología, causas y los diferentes tratamientos que hoy en día se tienen y en los que prima la utilización de la monoterapia con fármacos que produzcan remisión de la epilepsia con el menor número de eventos adversos y de interacciones clínicas.

Gabriela Ugarnes
Ciudad de Buenos Aires, Argentina

     

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  CLINICAL FEATURES AND MRI CHANGES IN DRUG-NAÏVE NEUROLOGIC WILSON’S DISEASE
Dystonia, dysarthria and tremor are common neurological features of neurologic Wilson’s disease. Abnormalities in MRI of brain are observed in all patients and these abnormalities are positively correlated with disease severity. The diffusion restriction is inversely correlated with the disease duration. MR Spectroscopy is also a sensitive tool for the diagnosis.

Satyabrata Pulai
Kolkata, In

  

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  EPILEPSY AND VITAMIN D
Vitamin D has a central role in the nervous system. Epilepsy patients generally exhibit low level of vitamin D and co-morbidities that are also related to vitamin D. Most antiepileptic drugs have an effect to lower serum level of vitamin D while vitamin D supplementation has been shown to decrease seizures. We recommend routine screening of epilepsy patients on antiepileptic drugs and supplement vitamin D if needed.

Zsófia Clemens
Budapest, Hu

  

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  ENDOSCOPIC ANTERIOR DECOMPRESSION IN CERVICAL DISC DISEASE
Endoscopic microforaminotomy is a safe and an effective alternative to microscopic anterior discectomy with or without fusion in the management of cervical disc disease.

Yad Ram Yadav
Jabalpur, In

     

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  ALTERED SUBCELLULAR DISTRIBUTION OF DISC1, CAMP LEVELS, AND NEURONAL MIGRATION IN SCHIZOPHRENIA AND BIPOLAR DISORDER
DISC1 (disrupted-in-schizophrenia-1) is considered a genetic risk factor for neuropsychiatric disorders. In this study, analysis in human olfactory neuronal precursors of schizophrenia and bipolar disorder patients showed decreased DISC1 levels in the cytoplasm, cAMP accumulation and diminished cell migration. The results suggest that these disorders share common molecular pathways that could hinder the neurodevelopmental process.

Jesús Muñoz-Estrada
México, México

     

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