DIAGNOSTICO DE LAS CAUSAS CURABLES DE ALDOSTERONISMO PRIMARIO




Artículos relacionadosArtículos relacionadosArtículos relacionados
Artículos afines de siicsalud publicados en los últimos 4 meses
AMLODIPINA EN EL TRATAMIENTO ACTUAL DE LA HIPERTENSIÓN
Journal of Clinical Hypertension 25(9):801-807
Difundido en siicsalud: 22 feb 2024
FACTORES PREDICTIVOS DE LOS TRASTORNOS HIPERTENSIVOS DEL EMBARAZO
Revista Latinoamericana de Hipertensión 18(3):1-4
Difundido en siicsalud: 2 feb 2024

DIAGNOSTICO DE LAS CAUSAS CURABLES DE ALDOSTERONISMO PRIMARIO

(especial para SIIC © Derechos reservados)
La prevalencia real de aldosteronismo primario es superior a la descrita con anterioridad. El diagnóstico precoz mediante técnicas adecuadas puede asociarse con la curación de la hipertensión y la prevención de eventos cardiovasculares.
Autor:
Gian Paolo F Rossi
Columnista Experto de SIIC

Institución:
University Hospital


Artículos publicados por Gian Paolo F Rossi
Coautores
Teresa Maria Seccia* Diego Miotto* 
University Hospital, Padua, Italia*
Recepción del artículo
12 de Agosto, 2009
Aprobación
3 de Junio, 2010
Primera edición
2 de Noviembre, 2010
Segunda edición, ampliada y corregida
7 de Junio, 2021

Resumen
La identificación de las formas quirúrgicamente corregibles de aldosteronismo primario (AP) requiere implementar la medición en sangre venosa suprarrenal (M-SVS) para demostrar la lateralización del exceso de aldosterona. En los pacientes con AP y secreción lateralizada de aldosterona, la suprarrenalectomía puede permitir la normalización de la presión arterial a largo plazo, así como la corrección del AP. En esta presentación resumimos los criterios para la selección de los pacientes candidatos a la M-SVS, la técnica para su realización y los parámetros para el análisis e interpretación de sus resultados.

Palabras clave
aldosterona, hipertensión, glándula suprarrenal, aldosteronismo primario, adenoma


Artículo completo

(castellano)
Extensión:  +/-13.15 páginas impresas en papel A4
Exclusivo para suscriptores/assinantes

Abstract
The identification of the surgically correctable forms of primary aldosteronism (PA) requires the adoption of the adrenal vein sampling (AVS) to demonstrate lateralized aldosterone excess. In the patients with PA and lateralized aldosterone secretion, adrenalectomy can provide long-term normalization of blood pressure and correction of PA. We herein summarize the criteria for selecting patients that will undergo AVS, the technique for performing AVS, and the criteria for analyzing and interpreting the results.

Key words
aldosterone, hypertension, adrenal gland, primary aldosteronism, adenoma


Full text
(english)
para suscriptores/ assinantes

Clasificación en siicsalud
Artículos originales > Expertos del Mundo >
página   www.siicsalud.com/des/expertocompleto.php/

Especialidades
Principal: Cardiología, Endocrinología y Metabolismo
Relacionadas: Anatomía Patológica, Bioquímica, Cirugía, Diagnóstico por Imágenes, Diagnóstico por Laboratorio, Genética Humana, Medicina Familiar, Medicina Interna



Comprar este artículo
Extensión: 13.15 páginas impresas en papel A4

file05.gif (1491 bytes) Artículos seleccionados para su compra



Enviar correspondencia a:
Gian Paolo F. Rossi, University Hospital DMCS -Clinica Medica 4 , 35126, via Giustiniani, 2, Padua, Italia
Bibliografía del artículo


1. Rossi GP. New concepts in adrenal vein sampling for aldosterone in the diagnosis of primary aldosteronism. Curr Hypertens Rep 9:90-97, 2007.
2. Rossi GP, Sechi LA, Giacchetti G, Ronconi V, Strazzullo P, Funder JW. Primary aldosteronism: cardiovascular, renal and metabolic implications. Trends Endocrinol Metab 19:88-90, 2008.
3. Rossi GP, Bernini G, Caliumi C, Desideri G, Fabris B, Ferri C, Ganzaroli C, Giacchetti G, Letizia C, Maccario M, Mallamaci F, Mannelli M, Mattarello MJ, Moretti A, Palumbo G, Parenti G, Porteri E, Semplicini A, Rizzoni D, Rossi E, Boscaro M, Pessina AC, Mantero F, PAPY Study Investigators. A prospective study of the prevalence of primary aldosteronism in 1,125 hypertensive patients. J Am Coll Cardiol 48:2293-2300, 2006.
4. Rayner BL, Opie LH, Davidson JS. The aldosterone/renin ratio as a screening test for primary aldosteronism. S Afr Med J 90:394-400, 2000.
5. Rossi E, Regolisti G, Negro A, Sani C, Davoli S, Perazzoli F. High prevalence of primary aldosteronism using postcaptopril plasma aldosterone to renin ratio as a screening test among Italian hypertensives. Am J Hypertens 15:896-902, 2002.
6. Schwartz GL, Chapman AB, Boerwinkle E, Kisabeth RM, Turner ST. Screening for primary aldosteronism: implications of an increased plasma aldosterone/renin ratio. Clin Chem 48:1919-1923, 2002.
7. Rossi GP, Rossi E, Pavan E, Rosati N, Zecchel R, Semplicini A, Perazzoli F, Pessina AC. Screening for primary aldosteronism with a logistic multivariate discriminant analysis. Clin Endocrinol (Oxf) 49:713-723, 1998.
8. Funder JW, Carey RM, Fardella C, Gomez-Sanchez CE, Mantero F, Stowasser M, Young WF,Jr, Montori VM, Endocrine Society. Case detection, diagnosis, and treatment of patients with primary aldosteronism: an endocrine society clinical practice guideline. J Clin Endocrinol Metab 93:3266-3281, 2008.
9. Rossi GP. Surgically correctable hypertension caused by primary aldosteronism. Best Pract Res Clin Endocrinol Metab 20:385-400, 2006.
10. Stowasser M, Klemm SA, Tunny TJ, Storie WJ, Rutherford JC, Gordon RD. Response to unilateral adrenalectomy for aldosterone-producing adenoma: effect of potassium levels and angiotensin responsiveness. Clin Exp Pharmacol Physiol 21:319-322, 1994.
11. Omura M, Sasano H, Fujiwara T, Yamaguchi K, Nishikawa T. Unique cases of unilateral hyperaldosteronemia due to multiple adrenocortical micronodules, which can only be detected by selective adrenal venous sampling. Metabolism 51:350-355, 2002.
12. Morioka M, Kobayashi T, Sone A, Furukawa Y, Tanaka H. Primary aldosteronism due to unilateral adrenal hyperplasia: report of two cases and review of the literature. Endocr J 47:443-449, 2000.
13. Katayama Y, Takata N, Tamura T, Yamamoto A, Hirata F, Yasuda H, Matsukuma S, Daido Y, Sasano H. A case of primary aldosteronism due to unilateral adrenal hyperplasia. Hypertens Res 28:379-384, 2005.
14. Mulatero P, Dluhy RG, Giacchetti G, Boscaro M, Veglio F, Stewart PM. Diagnosis of primary aldosteronism: from screening to subtype differentiation. Trends Endocrinol Metab 16:114-119, 2005.
15. Rossi GP, Ganzaroli C, Miotto D, De Toni R, Palumbo G, Feltrin GP, Mantero F, Pessina AC. Dynamic testing with high-dose adrenocorticotrophic hormone does not improve lateralization of aldosterone oversecretion in primary aldosteronism patients. J Hypertens 24:371-379, 2006.
16. Fallo F, Barzon L, Boscaro M, Sonino N. Coexistence of aldosteronoma and contralateral nonfunctioning adrenal adenoma in primary aldosteronism. Am J Hypertens 10:476-478, 1997.
17. Magill SB, Raff H, Shaker JL, Brickner RC, Knechtges TE, Kehoe ME, Findling JW. Comparison of adrenal vein sampling and computed tomography in the differentiation of primary aldosteronism. J Clin Endocrinol Metab 86:1066-1071, 2001.
18. Kloos RT, Gross MD, Francis IR, Korobkin M, Shapiro B. Incidentally discovered adrenal masses. Endocr Rev 16:460-484, 1995.
19. Mantero F, Terzolo M, Arnaldi G, Osella G, Masini AM, Ali A, Giovagnetti M, Opocher G, Angeli A. A survey on adrenal incidentaloma in Italy. Study Group on Adrenal Tumors of the Italian Society of Endocrinology. J Clin Endocrinol Metab 85:637-644, 2000.
20. Young WF,Jr. Clinical practice. The incidentally discovered adrenal mass. N Engl J Med 356:601-610, 2007.
21. Blumenfeld JD, Sealey JE, Schlussel Y, Vaughan ED,Jr, Sos TA, Atlas SA, Muller FB, Acevedo R, Ulick S, Laragh JH. Diagnosis and treatment of primary hyperaldosteronism. Ann Intern Med 121:877-885, 1994.
22. Stowasser M, Gordon RD, Rutherford JC, Nikwan NZ, Daunt N, Slater GJ. Diagnosis and management of primary aldosteronism. J Renin Angiotensin Aldosterone Syst 2:156-169, 2001.
23. Young WF,Jr. Primary aldosteronism: management issues. Ann N Y Acad Sci 970:61-76, 2002.
24. Rossi GP, Chiesura-Corona M, Tregnaghi A, Zanin L, Perale R, Soattin S, Pelizzo MR, Feltrin GP, Pessina AC. Imaging of aldosterone-secreting adenomas: a prospective comparison of computed tomography and magnetic resonance imaging in 27 patients with suspected primary aldosteronism. J Hum Hypertens 7:357-363, 1993.
25. Lockhart ME, Smith JK, Kenney PJ. Imaging of adrenal masses. Eur J Radiol 41:95-112, 2002.
26. Gordon RD. Primary aldosteronism. J Endocrinol Invest 18:495-511, 1995.
27. Young WF Jr, Stanson AW, Grant CS, Thompson GB, Van Heerden JA. Primary aldosteronism: adrenal venous sampling. Surgery 120:913-9; discussion 919-20, 1996.
28. Phillips JL, Walther MM, Pezzullo JC, Rayford W, Choyke PL, Berman AA, Linehan WM, Doppman JL, Gill JR Jr. Predictive value of preoperative tests in discriminating bilateral adrenal hyperplasia from an aldosterone-producing adrenal adenoma. J Clin Endocrinol Metab 85:4526-4533, 2000.
29. Rossi GP, Sacchetto A, Chiesura-Corona M, De Toni R, Gallina M, Feltrin GP, Pessina AC. Identification of the etiology of primary aldosteronism with adrenal vein sampling in patients with equivocal computed tomography and magnetic resonance findings: results in 104 consecutive cases. J Clin Endocrinol Metab 86:1083-1090, 2001.
30. Young WF, Stanson AW, Thompson GB, Grant CS, Farley DR, Van Heerden JA. Role for adrenal venous sampling in primary aldosteronism. Surgery 136:1227-1235, 2004.
31. Stowasser M, Gordon RD, Rutherford JC, Nikwan NZ, Daunt N, Slater GJ. Diagnosis and management of primary aldosteronism. J Renin Angiotensin Aldosterone Syst 2:156-169, 2001.
32. Daunt N. Adrenal vein sampling: how to make it quick, easy, and successful. Radiographics 25(Suppl.1):S143-58, 2005.
33. Espiner EA, Ross DG, Yandle TG, Richards AM, Hunt PJ. Predicting surgically remedial primary aldosteronism: role of adrenal scanning, posture testing, and adrenal vein sampling. J Clin Endocrinol Metab 88:3637-3644, 2003.
34. Rossi GP, Pitter G, Bernante P, Motta R, Feltrin G, Miotto D. Adrenal vein sampling for primary aldosteronism: the assessment of selectivity and lateralization of aldosterone excess baseline and after adrenocorticotropic hormone (ACTH) stimulation. J Hypertens 26:989-997, 2008.
35. Rossi GP, Belfiore A, Bernini G, Desideri G, Fabris B, Ferri C, Giacchetti G, Letizia C, Maccario M, Mallamaci F, Mannelli M, Palumbo G, Rizzoni D, Rossi E, Agabiti-Rosei E, Pessina AC, Mantero F, Primary Aldosteronism Prevalence in Italy Study Investigators. Comparison of the captopril and the saline infusion test for excluding aldosterone-producing adenoma. Hypertension 50:424-431, 2007.
36. Rossi GP, Pitter G, Miotto D. To stimulate or not to stimulate: is adrenocorticotrophic hormone testing necessary, or not? J Hypertens 25:481, 2007.
37. Seccia TM, Miotto D, De Toni R, Pitter G, Mantero F, Pessina AC, Rossi GP. Adrenocorticotropic hormone stimulation during adrenal vein sampling for identifying surgically curable subtypes of primary aldosteronism: comparison of 3 different protocols. Hypertension 53:761-766, 2009.
38. Mansoor GA, Malchoff CD, Arici MH, Karimeddini MK, Whalen GF. Unilateral adrenal hyperplasia causing primary aldosteronism: limitations of I-131 norcholesterol scanning. Am J Hypertens 15:459-464, 2002.
39. Bossuyt PM, Reitsma JB, Bruns DE, Gatsonis CA, Glasziou PP, Irwig LM, Moher D, Rennie D, de Vet HC, Lijmer JG, Standards for Reporting of Diagnostic Accuracy. The STARD statement for reporting studies of diagnostic accuracy: explanation and elaboration. Clin Chem 49:7-18, 2003.
40. Diaz-Cano SJ, De Miguel M, Blanes A, Tashjian R, Galera H, Wolfe HJ. Clonality as expression of distinctive cell kinetics patterns in nodular hyperplasias and adenomas of the adrenal cortex. Am J Pathol 156:311-319, 2000.
41. Rossi GP, Vendraminelli R, Cesari M, Pessina AC. A thoracic mass with hypertension and hypokalaemia. Lancet 356:1570, 2000.
42. Seccia TM, Fassina A, Nussdorfer GG, Pessina AC, Rossi GP. Aldosterone-producing adrenocortical carcinoma: an unusual cause of Conn's syndrome with an ominous clinical course. Endocr Relat Cancer 12:149-159, 2005.
43. Sukor N, Gordon RD, Ku YK, Jones M, Stowasser M. Role of unilateral adrenalectomy in bilateral primary aldosteronism: a 22-year single center experience. J Clin Endocrinol Metab 94:2437-2445, 2009.
44. Wolf-Maier K, Cooper RS, Banegas JR, Giampaoli S, Hense HW, Joffres M, Kastarinen M, Poulter N, Primatesta P, Rodriguez-Artalejo F, Stegmayr B, Thamm M, Tuomilehto J, Vanuzzo D, Vescio F. Hypertension prevalence and blood pressure levels in 6 European countries, Canada, and the United States. JAMA 289:2363-2369, 2003.
45. Rundback JH, Sacks D, Kent KC, Cooper C, Jones D, Murphy T, Rosenfield K, White C, Bettmann M, Cortell S, Puschett J, Clair D, Cole P, AHA Councils on Cardiovascular Radiology, High Blood Pressure Research, Kidney in Cardiovascular Disease, Cardio-Thoracic and Vascular Surgery, and Clinical Cardiology, and the Society of Interventional Radiology FDA Device Forum Committee. American Heart Association. Guidelines for the reporting of renal artery revascularization in clinical trials. American Heart Association. Circulation 106:1572-1585, 2002.
46. Dluhy RG, Lifton RP. Glucocorticoid-remediable aldosteronism. J Clin Endocrinol Metab 84:4341-4344, 1999.
47. Mulatero P, Veglio F, Pilon C, Rabbia F, Zocchi C, Limone P, Boscaro M, Sonino N, Fallo F. Diagnosis of glucocorticoid-remediable aldosteronism in primary aldosteronism: aldosterone response to dexamethasone and long polymerase chain reaction for chimeric gene. J Clin Endocrinol Metab 83:2573-2575, 1998.
48. Pascoe L, Jeunemaitre X, Lebrethon MC, Curnow KM, Gomez-Sanchez CE, Gasc JM, Saez JM, Corvol P. Glucocorticoid-suppressible hyperaldosteronism and adrenal tumors occurring in a single French pedigree. J Clin Invest 96:2236-2246, 1995.

 
 
 
 
 
 
 
 
 
 
 
 
Está expresamente prohibida la redistribución y la redifusión de todo o parte de los contenidos de la Sociedad Iberoamericana de Información Científica (SIIC) S.A. sin previo y expreso consentimiento de SIIC.
Artículos relacionadosMás relacionadosAtículos relacionados
ua31618